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Indeed, during APAP-induced toxicity there is an increased generation of mitochondrial superoxide anion production, stimulated by impaired mitochondrial respiration and formation of adducts between the reactive metabolite NAPQI and sulfhydryls moieties from mitochondrial proteins (Yan et al., 2010)
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Gut 2007;56:982–90 [DOI] [PMC free article] [PubMed] [Google Scholar] 91.Latchoumycandane C, Goh CW, Ong MM, Boelsterli UA.Mitochondrial protection by the JNK inhibitor leflunomide rescues mice from acetaminophen-induced liver injury
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